Accès gratuit
Numéro |
Med Sci (Paris)
Volume 29, Numéro 12, Décembre 2013
|
|
---|---|---|
Page(s) | 1145 - 1150 | |
Section | M/S Revues | |
DOI | ||
Publié en ligne | 20 décembre 2013 |
- MacDonald TT, Monteleone I, Fantini MC, Monteleone G. Regulation of homeostasis and inflammation in the intestine. Gastroenterology 2011 ; 140 : 1768–1775.
- McGuckin MA, Eri R, Simms LA, et al. Intestinal barrier dysfunction in inflammatory bowel diseases. Inflamm Bowel Dis 2009 ; 15 : 100–113.
- Maloy KJ, Powrie F. Intestinal homeostasis and its breakdown in inflammatory bowel disease. Nature 2011 ; 474 : 298–306.
- Bevins CL, Salzman NH. Panethcells, antimicrobial peptides and maintenance of intestinal homeostasis. Nat Rev Microbiol 2011 ; 9 : 356–368.
- Ramasundara M, Leach ST, Lemberg DA, Day AS. Defensins and inflammation: the role of defensins in inflammatory bowel disease. J Gastroenterol Hepatol 2009 ; 24 : 202–208.
- Ganz T. Defensins: antimicrobial peptides of innate immunity. Nat Rev Immunol 2003 ; 3 : 710–720.
- Roda G, Sartini A, Zambon E, et al. Intestinal epithelial cells in inflammatory bowel diseases. World J Gastroenterol 2010 ; 16 : 4264–4271.
- Goto Y, Ivanov I. Intestinal epithelial cells as mediators of the commensal-host immune crosstalk. Immunol Cell Biol 2013 ; 91 : 204–214.
- Laukoetter MG, Bruewer M, Nusrat A. Regulation of the intestinal epithelial barrier by the apical junctional complex. Curr Opin Gastroenterol 2006 ; 22 : 85–89.
- Assimakopoulos SF, Papageorgiou I, Charonis A. Enterocyte’s tight junctions: from molecules to diseases. World J Gastrointest Pathophysiol 2011 ; 15 : 123–137.
- Sudha PS, Devaraj H, Devaraj N. Adherence of Shigella dysenteriae 1 to human colonic mucin. Curr Microbiol 2001 ; 42 : 381–387.
- Vaishnava S, Behrendt CL, Ismail AS, et al. Paneth cells directly sense gut commensals and maintain homeostasis at the intestinal host-microbial interface. Proc Natl Acad Sci USA 2008 ; 105 : 20858–20863.
- Slack E. Innate and adaptive immunity cooperate flexibly to maintain host-microbiota mutualism. Science 2009 ; 325 : 617–620.
- Yeretssian G. Effector functions of NLRs in the intestine: innate sensing, cell death, and disease. Immunol Res 2012 ; 54 : 25–36.
- Lee J, Mo JH, Shen C, et al. Toll-like receptor signaling in intestinal epithelial cells contributes to colonic homoeostasis. Curr Opin Gastroenterol 2007 ; 23 : 27–31.
- Kaser A, Niederreiter L, Blumberg RS. Genetically determined epithelial dysfunction and its consequences for microflora host interactions. Cell Mol Life Sci 2011 ; 68 : 3643–3649.
- Thjodleifsson B, Sigthorsson G, Cariglia N, et al. Subclinical intestinal inflammation: an inherited abnormality in Crohn’s disease relatives? Gastroenterology 2003 ; 124 : 1728–1737.
- Breslin NP, Nash C, Hilsden RJ, et al. Intestinal permeability is increased in a proportion of spouses of patients with Crohn’s disease. Am J Gastroenterol 2001 ; 96 : 2934–2938.
- Utech M, Mennigen R, Bruewer M., Endocytosis and recycling of tight junction proteins in inflammation. J Biomed Biotechnol 2010 ; 2010 : 484987.
- Kosiewicz MM, Nast CC, Krishnan A, et al. Th1-type responses mediate spontaneous ileitis in a novel murine model of Crohn’s disease. J Clin Invest 2001 ; 107 :695–702.
- Vermeire S, Rutgeerts P. Current status of genetics research in inflammatory bowel disease. Genes Immun 2005 ; 6 : 637–645.
- Kanoh A, Takeuchi H, Kato K, et al. Interleukin-4 induces specific pp-GalNAc-T expression and alterations in mucin O-glycosylation in colonic epithelial cells. Biochim Biophys Acta 2008 ; 1780 :577–584.
- Lu P, Burger-van Paassen N, Van der Sluis M, et al. Colonic gene expression patterns of mucin Muc2 knockout mice reveal various phases in colitis development. Inflamm Bowel Dis 2011 ; 17 : 2045–2057.
- Tytgat KMA, van der Wal JWG, Einerhand AWC, et al. Quantitative analysis of MUC2 synthesis in ulcerative colitis. Biochem Biophys Res Commun 1996 ; 224 : 397–405.
- Mashimo H, Wu DC, Podolsky DK, Fishman MC. Impaired defense of intestinal mucosa in mice lacking intestinal trefoil factor. Science 1996 ; 274 : 262–265.
- Tomasetto C, Masson R, Linares J, et al. pS2/TFF1 interacts directly with the VWFC cysteine-rich domains of mucins. Gastroenterology 2000 ; 118 : 70–80.
- Zhao F, Edwards R, Dizon D, et al. Disruption of Paneth and goblet cell homeostasis and increased endoplasmic reticulum stress in Agr2-/- mice. Dev Biol 2010 ; 338 : 270–279.
- Kaser A, Martinez-Naves E, Blumberg RS. Endoplasmic reticulum stress: implications for inflammatory bowel disease pathogenesis. Curr Opin Gastroenterol 2010 ; 26 : 318–326.
- Tréton X, Pedruzzi E, Cazals-Hatem D, et al. Altered endoplasmic reticulum stress affects translation in inactive colon tissue from patients with ulcerative colitis. Gastroenterology 2011 ; 141 : 1024–1035.
- Kaser A. XBP1 links ER stress to intestinal inflammation and confers genetic risk for human inflammatory bowel disease. Cell 2008 ; 134 : 743–756.
- Wehkamp J, Harder J, Weichenthal M, et al. NOD2 (CARD15) mutations in Crohn’s disease are associated with diminished mucosal alpha defensin expression. Gut 2004 ; 53 : 1653–1664.
- Harris G, KuoLee R, Chen W. Role of Toll-like receptors in health and diseases of gastrointestinal tract. World J Gastroenterol 2006 ; 12 : 2149–2160.
- Cadwell, K. A key role for autophagy and the autophagy gene Atg16l1 in mouse and human intestinal Paneth cells. Nature 2008 ; 456 : 259–263.
- Ayabe T, Satchell DP, Wilson CL, et al. Secretion of microbicidal alpha-defensins by intestinal Paneth cells in response to bacteria. Nat Immunol 2000 ; 1 : 113–118.
- Arnott ID, Ho GT, Nimmo ER, Satsangi J., Toll-like receptor 4 gene in IBD: further evidence for genetic heterogeneity in Europe. Gut 2005 ; 54 : 308.
- Hiemstra IH, Bouma G, Geerts D, et al. Nod2 improves barrier function of intestinal epithelial cells via enhancement of TLR responses. Mol Immunol 2012 ; 52 : 264–272.
- Jamilloux Y, Henry T. Les inflammasomes : plates-formes de l’immunité innée. Med Sci (Paris) 2013 ; 29 : 975–984. [EDP Sciences]
- Matricon J. Immunopathogenèse des maladies inflammatoires chroniques de l’intestin. Med Sci (Paris) 2010 ; 26 : 405–410. [CrossRef] [EDP Sciences] [PubMed]
- Normand S, Secher T, Chamaillard M. La dysbiose, une nouvelle entité en médecine. Med Sci (Paris) 2013 ; 29 : 586–589. [EDP Sciences]
- Rio MC. Fonctions paradoxales pour TFF1. Med Sci (Paris) 2002 ; 18 : 1177–1178. [CrossRef] [EDP Sciences]
- Zahraoui A. Les jonctions serrées : plate-forme de régulation de la prolifération et de la polarité cellulaire. Med Sci (Paris) 2004 ; 20 : 580–585. [CrossRef] [EDP Sciences] [PubMed]
Les statistiques affichées correspondent au cumul d'une part des vues des résumés de l'article et d'autre part des vues et téléchargements de l'article plein-texte (PDF, Full-HTML, ePub... selon les formats disponibles) sur la platefome Vision4Press.
Les statistiques sont disponibles avec un délai de 48 à 96 heures et sont mises à jour quotidiennement en semaine.
Le chargement des statistiques peut être long.